MIT School of Science announces 2023 Infinite Mile Awards
Seven staff members honored for their dedication to the School of Science and to the Institute.
Seven staff members honored for their dedication to the School of Science and to the Institute.
A new study tests an alternative to external stimulation for measuring when subjects lose and regain responsiveness during sedation and anesthesia.
Neurons that form part of a memory circuit are among the first brain cells to show signs of neurodegeneration in Alzheimer’s disease.
The peptide blocks a hyperactive brain enzyme that contributes to the neurodegeneration seen in Alzheimer’s and other diseases.
When astrocyte function is disrupted, neurons in the brain’s motor cortex struggle to execute and refine motion, a new study in mice shows.
The brain applies rhythms to physical patches of the cortex to selectively control just the right neurons at the right times to do the right things.
On March 10 the FDA approved Trofinetide, a drug based on the protein IGF-1. The MIT professor's original research showing that IGF-1 could treat Rett was published in 2009.
Seven postdocs and research scientists honored for contributions to the Institute.
First detailed mapping and modeling of thalamus inputs onto visual cortex neurons show brain leverages “wisdom of the crowd” to process sensory information.
“Single-cell profiling” is helping neuroscientists see how disease affects major brain cell types and identify common, potentially targetable pathways.
Comparing models of working memory with real-world data, MIT researchers find information resides not in persistent neural activity, but in the pattern of its connections.
Longtime MIT professor of neuroscience led research behind 200 patents, laying the groundwork for numerous medical products.
MIT researchers report early-stage clinical study results of tests with noninvasive 40-hertz light and sound treatment.
In people carrying APOE4, a key brain cell mismanages cholesterol needed to insulate neurons properly — another sign APOE4 contributes to disease by disrupting brain lipids.
Scientists hypothesize that, as in a hibernating turtle, the brain under sedation and deprived of oxygen may assume a protective state.