Researchers and staff from MIT, including from the Simons Center for the Social Brain, collaborated with schoolchildren with special needs to create art, have fun, and learn from each other.
With support from The Marcus Foundation, an MIT neuroscientist and a Harvard Medical School immunologist will study the “fever effect” in an effort to devise therapies that mimic its beneficial effects.
Team-based targeted projects, multi-mentor fellowships ensure that scientists studying social cognition, behavior, and autism integrate multiple perspectives and approaches to pressing questions.
Award recognizes professor's synaptic plasticity research, its translation to potential amblyopia and autism treatments, and his career of mentorship.
Thirteen new graduate student fellows will pursue exciting new paths of knowledge and discovery.
MIT engineers’ new technology can probe the neural circuits that influence hunger, mood, and a variety of diseases.
Faulty versions of the Foxp2 gene disrupt neurons’ ability to form synapses in brain regions involved in speech, a new study shows.
Students working in Boston Medical Center’s Autism Program make an impact while experiencing the whirlwind atmosphere of a hospital.
At an exhibition marking two decades since a transformative gift from the Picower Foundation, current and alumni members described research at the forefront of neuroscience and beyond.
Gloria Choi’s studies of how the immune system and nervous system influence each other could yield new approaches to treating neurological disorders.
A commonly used screening test creates a gender gap that may hinder diagnosis and treatment for women and girls.
Using organoids to model early development, researchers used an emerging microscopy technology to see that new neurons struggled to reach their developmental destination.
A new computational model could explain differences in recognizing facial emotions.
K. Lisa Yang Brain-Body Center to investigate the brain’s complex relationship with other body systems.
Infection during pregnancy with elevated levels of the cytokine IL-17a may yield microbiome alterations that prime offspring for aberrant immune responses, mouse study suggests.